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Scientist at Yale School of Medicine have discovered that the brain’s appetite area uses fat fuel by adding oxidative free radicals”molecules that have been linked to aging and Nero degeneration.

The results were published in the journal Nature, and hint that antioxidants could be a major player in regards to weight control

The documents chief authors were Sabrina Diano and Tamas Horvath who are two associate professor and professor, at the Building of Obstetrics, Gynecology & Reproductive Sciences and Neurobiology.

The researchers are also members of the Section of Comparative Medicine

The study found unlike the widely held belief that the brain does not use fat as fuel, the research proved otherwise.

This means that if you disrupt the free radicals, you may affect eating and digestion process.

The results also showed that every time a sensation of fullness or satiety is achieved during a meal, you may be eating away some a person’s maximum lifespan.

The study also noted that free radical production occured when thre brain cells were active.

The researchers used mice to conduct and determine how the human brain uses neuronal activation.

This response is determined by Gherlein which is a hormone produced in the stomach.

This hormone is also associated with:

• growth hormone release
• learning

The scientist found that the hormone produced a appetite by placing fat in the hypothalamic mitochondria.

This compound produces harmful free radicals which are scavenged by a mitochondrial protein known as uncoupling protein 2 (UCP2).

The study shows that timing is everything when taking antioxidants to control appetite.

An increase in appetite tends to occur when antioxidants are taken on a empty stomach yet when consumed with food they tend to regulate.

Further analysis is needed to ascertain whether any regiment of antioxidants taken orally could be used to affect appetite in animals and humans.